Publications
Knowledge generated at IrsiCaixa is shared with the scientific community through the publication of articles in high impact scientific journals.
Trans-infection but not infection from within endosomal compartments after cell-to-cell HIV-1 transfer to CD4+ T cells.
Antiretroviral agents effectively block HIV replication after cell-to-cell transfer.
β5 integrin is the major contributor to the αVintegrin-mediated blockade of HIV-1 replication.
Development of resistance to VIR-353 with cross-resistance to the natural HIV-1 entry virus inhibitory peptide (VIRIP).
Compensatory mutations rescue the virus replicative capacity of VIRIP-resistant HIV-1.
ADS-J1 inhibits HIV-1 entry by interacting with gp120 and does not block fusion-active gp41 core formation.
ZNRD1 (zinc ribbon domain-containing 1) is a host cellular factor that influences HIV-1 replication and disease progression.
Endocytosis of HIV: anything goes.
Different selection patterns of resistance and cross-resistance to HIV-1 agents targeting CCR5.
Deletion of the late cornified envelope LCE3B and LCE3C genes as a susceptibility factor for psoriasis.